RVA-induced necrosome formation consisting of RIPK1, RIPK3, and MLKL
Por um escritor misterioso
Descrição
RVA-induced necrosome formation consisting of RIPK1, RIPK3, and MLKL
MLKL in cancer: more than a necroptosis regulator. - Abstract - Europe PMC
MLKL in cancer: more than a necroptosis regulator. - Abstract - Europe PMC
Opposite Effects of Apoptotic and Necroptotic Cellular Pathways on Rotavirus Replication
Transient knockdown of components along the RIPK1–RIPK3–MLKL–PGAM5–Drp1
IJMS, Free Full-Text
Cardiomyocyte Alpha-1A Adrenergic Receptors Mitigate Postinfarct Remodeling and Mortality by Constraining Necroptosis
RIPK3-and MLKL-dependent necroptosis mediated by various stimuli.
Necroptosis molecular mechanisms: Recent findings regarding novel necroptosis regulators
From (Tool)Bench to Bedside: The Potential of Necroptosis Inhibitors
Overview of necroptotic signaling pathway. Necroptosis can be induced
RIPK3 signaling and its role in the pathogenesis of cancers
Frontiers Convergent Loss of the Necroptosis Pathway in Disparate Mammalian Lineages Shapes Viruses Countermeasures
Necroptosis: a regulated inflammatory mode of cell death, Journal of Neuroinflammation
Frontiers Exploiting Necroptosis for Therapy of Acute Lymphoblastic Leukemia