Editing one gene extends mouse life expectancy by 23%

Por um escritor misterioso

Descrição

By modifying just one gene, researchers were able to extend the life expectancy of mice by 23%, and they think their results may translate to humans.
Editing one gene extends mouse life expectancy by 23%
Base Editing in Progeria
Editing one gene extends mouse life expectancy by 23%
Exercise preserves physical fitness during aging through AMPK and mitochondrial dynamics
Editing one gene extends mouse life expectancy by 23%
TALEN-Mediated Gene Editing of HBG in Human Hematopoietic Stem Cells Leads to Therapeutic Fetal Hemoglobin Induction: Molecular Therapy - Methods & Clinical Development
Editing one gene extends mouse life expectancy by 23%
Improved health-span and lifespan in mtDNA mutator mice treated with the mitochondrially targeted antioxidant SkQ1
Editing one gene extends mouse life expectancy by 23%
Dietary compounds which successfully extended lifespan in the National
Editing one gene extends mouse life expectancy by 23%
CRISPR doubles lifespan of mice with rapid ageing disease progeria
Editing one gene extends mouse life expectancy by 23%
Genome editing in the mouse brain with minimally immunogenic Cas9 RNPs: Molecular Therapy
Editing one gene extends mouse life expectancy by 23%
Programming large target genomic deletion and concurrent insertion via a prime editing-based method: PEDAR
Editing one gene extends mouse life expectancy by 23%
Dose imbalance of DYRK1A kinase causes systemic progeroid status in Down syndrome by increasing the un-repaired DNA damage and reducing LaminB1 levels - eBioMedicine
Editing one gene extends mouse life expectancy by 23%
In vivo somatic cell base editing and prime editing - ScienceDirect
Editing one gene extends mouse life expectancy by 23%
IJMS, Free Full-Text
Editing one gene extends mouse life expectancy by 23%
CRISPR/Cas9-Mediated miR-29b Editing as a Treatment of Different Types of Muscle Atrophy in Mice - ScienceDirect
Editing one gene extends mouse life expectancy by 23%
Dose imbalance of DYRK1A kinase causes systemic progeroid status in Down syndrome by increasing the un-repaired DNA damage and reducing LaminB1 levels - eBioMedicine
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